Vitiligo or Leucoderma or White skin

It is a gained idiopathic depigmentary condition, which however worldwide in circulation, generally basic in India and other tropical nations. It affects all age groups and both sexes.

  1. Exact causation is not known, hereditary predisposition can't be ruled out
  2. Dietary deficiency of proteins and cuprominerals is a major factor in our cases
  3. Chronic amoebiasis, chronic dyspepsia, intestinal worms may be additional factors
  4. Trauma, local irritation
  5. Acute stress, an endocrinal disorder associated with diabetes, pernicious anemia, thyrotoxicosis, myxedema, and Addison's auto-immunity

  1. A defect in the enzyme tyrosine hydroxylase is held responsible for vitiligo. According to somebody, it is trophoneurosis and melatonin, a substance secreted at nerve endings inhibits tyrosinase, thus interfering in pigment formation. DOPA staining shows that melanocytes are absent. In dynamic cases, mononuclear embracing at the cellar layer is a conspicuous component.
  2. Patches enlarge slowly and may affect the whole body, face, dorsum of fingers and hands, waist, and legs.
  3. Hair may or may not become depigmented
  4. The patient's skin is susceptible to even minor trauma, heals with depigmentation

Signs / Symptoms
  1. Totally depigmented macules patches of differing sizes and shapes. The early lesion may be pale white and ill-defined.
  2. The hemoglobin content of the blood is low and sometimes intestinal parasites and infections can be detected. Patients complain of easy fatigability, may be anemic.

It is usually apparent. In doubtful and early cases, Wood's lamp is of great help in diagnosis. Vitiligo areas are milky white while others lack this milky white coloration.

  1. The patient and the relatives should be assured about its non-infectious and non-hereditary maturity and that it has no relationship to leprosy whatsoever.
  2. The patient should be instructed to avoid physical trauma, board-spectrum antibiotics
  3. Control of etiological factors
  4. Diet: Cheese, butter-mild, almonds, figs, germinating grains(which are rich in tyrosinase), and bale fruit(syrup, squash, or preserved) are added with benefit
  5. U.V.R in stabilized cases
  6. Ultraviolet rays of log range

Homeopathic approach!

Arsenicum album 200 is the great medicine for this disease

Ars sulph flavum 1M another good medicine

Zinc Phosphate 6 is a good medicine

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